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Background: Alcohol drinking affects atherosclerotic progression mainly through blood pressure and lipid metabolism. The purpose of the present study was to clarify whether effects of alcohol drinking on atherosclerotic risk factors differ by gender and age.
Background: Alcoholic beverages stimulate gastric acid secretion and increase the appetite. Although ingested ethanol stimulates pancreatic secretion, alcoholic beverages contain several congeners. N‐methyltyramine (NMT) was isolated from beer as a factor in stimulating gastric acid secretion....
Background: Excessive consumption of alcohol is involved in the onset of pancreatitis. However, most of heavy drinkers do not always develop chronic pancreatitis. Various genetic factors appear to be involved in these individual differences in onset of chronic alcoholic pancreatitis. Here we...
Background: We previously reported that an NAD‐dependent in situ retinoic acid supply system, which comprises some isoforms of alcohol dehydrogenase (ADH) and aldehyde dehydrogenase (ALDH) and provides retinoic acid from retinol via a 2‐step oxidation process, exists in the rat esophagus....
Background: Pathologists examining victims of sudden unexpected death encounter alcoholics more often than expected; alcohol may play a role in sudden arrhythmic death. Here we determine whether a pattern of alcohol consumption, chronic ethanol intake, and withdrawal increases the incidence of...
Background: The pathogenesis of nonalcoholic steatohepatitis (NASH) is still unclear. Recently, the 2‐hit hypothesis was proposed, in which nitric oxide production, representing oxidative stress, was proposed as a very important candidate for the second hit.
Background/Aims: Xenon computed tomography (Xe‐CT) is a noninvasive method of quantifying and visualizing tissue blood flow (TBF). For the liver, Xe‐CT allows separate measurement of hepatic arterial and portal venous TBF. The present study evaluated the usefulness of Xe‐CT as a noninvasive...
Background: Swift increase in alcohol metabolism (SIAM) is usually evoked by a large dose of ethanol, which is often demonstrated by an abrupt increase in oxygen uptake. SIAM was induced by low doses of ethanol and evaluated by pharmacokinetic analyses of ethanol and its metabolites.
Background: Deficiency of ADAMTS13 (adisintegrin‐like and metalloproteinase with thrombospondin type‐1 motifs 13) results in an increase in unusually large von Willebrand factor multimer (UL‐VWFM) of the plasma and finally causes microcirculatory disturbance. Our previous study demonstrated that...
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