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Apoptosis of arterial cells induced by oxidized low–density
lipoprotein (oxLDL) is thought to contribute to the progression of vascular
dysfunction and atherogenesis. It is well established that diabetes mellitus is
accompanied by both glycosylation and oxidation LDL, but the biological
We tested the hypothesis that deletion of adenylyl cyclase type V
(ACV) would be associated with decreased left ventricular (LV) contractile
function and responsiveness to β–adrenergic receptor (βAR) stimulation.
Absence of cardiac ACV expression was confirmed by RT–PCR and
Pulmonary margination of neutrophils may contribute to lung
damage after extracorporeal circulation for cardiac surgery. We evaluated
single–pass pulmonary neutrophil kinetics using the multiple indicator–dilution
technique in control pigs (n = 10), after sternotomy alone (sterno, n =
ATP–sensitive potassium channels are opened during the course
of ischemic preconditioning (IP). As experimental data suggest that opening
of sarcolemmal ATP–sensitive potassium channels underlie ST elevation during
myocardial ischemia, one would expect to observe increased ST elevation
It is still unknown whether remote ischemic preconditioning is
mediated by a humoral or a neurogenic mechanism from the preconditioning
to the preconditioned tissue. The purpose of the following study was to
identify a possible humoral trigger of ischemic myocardial preconditioning
Acetylcholine (ACh) and opioid receptor agonists trigger the preconditioned
phenotype through sequential activation of the epidermal
growth factor (EGF) receptor, phosphatidylinositol 3-kinase (PI3-K), Akt,
and nitric oxide synthase (NOS), and opening of mitochondrial (mito) KATP
We aimed to assess the role of the nitric oxide (NO)–cGMP pathway
in cardioprotection by brief intermittent ischemias at the onset of reperfusion
(i.e., post–conditioning (Post–con)). We also evaluated the role of coronary
flow and pressure in Post–con. Rat isolated hearts perfused at constant–...
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