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Severe pneumonia by aciclovir-resistant varicella-zoster virus during etanercept therapy

Severe pneumonia by aciclovir-resistant varicella-zoster virus during etanercept therapy Letters to the Editor 4 Takahira M, Kawano M, Zen Y, Minato H, Yamada K, This case highlights the systemic nature of IgG4þ Sugiyama. IgG4-related chronic sclerosing dacryoadeni- MOLPS and the importance of distinguishing it from tis. Arch Ophthalmol 2007;125:1575–8. clinically similar entities such as SS. Clinical recognition 5 Aoki A, Sato K, Itabashi M et al. A case of Mikulicz’s of IgG4þ MOLPS is important, as CS may provide disease complicated with severe interstitial complete relief from symptoms and reduce morbidity, nephritis associated with IgG4. Clin Exp Nephrol 2009;13: particularly if instigated early in the course of the disease. 367–72. Comprehensive, validated diagnostic criteria and a 6 Miyake K, Moriyama M, Aizawa K et al. Peripheral greater understanding of the pathogenesis of IgG4þ CD4þ T cells showing a Th2 phenotype in a MOLPS are required. patient with Mikulicz’s disease associated with lymphadenopathy and pleural effusion. Mod Rheumatol 2008;18:86–90. Rheumatology key message 7 Zen Y, Fujii T, Harada K et al. Th2 and regulatory immune . CSSD-manifesting IgG4þ MOLPS can present with reactions are increased in immunoglobin G4-related SPH and remarkable IgG4-linked IL-4 elevation. sclerosing pancreatitis and cholangitis. Hepatology 2007; 45:1538–46. 8 Ueno K, Watanabe T, Kawata Y et http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Rheumatology Oxford University Press

Severe pneumonia by aciclovir-resistant varicella-zoster virus during etanercept therapy

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References (8)

Publisher
Oxford University Press
Copyright
The Author 2010. Published by Oxford University Press on behalf of the British Society for Rheumatology. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org
ISSN
1462-0324
eISSN
1462-0332
DOI
10.1093/rheumatology/keq110
pmid
20484341
Publisher site
See Article on Publisher Site

Abstract

Letters to the Editor 4 Takahira M, Kawano M, Zen Y, Minato H, Yamada K, This case highlights the systemic nature of IgG4þ Sugiyama. IgG4-related chronic sclerosing dacryoadeni- MOLPS and the importance of distinguishing it from tis. Arch Ophthalmol 2007;125:1575–8. clinically similar entities such as SS. Clinical recognition 5 Aoki A, Sato K, Itabashi M et al. A case of Mikulicz’s of IgG4þ MOLPS is important, as CS may provide disease complicated with severe interstitial complete relief from symptoms and reduce morbidity, nephritis associated with IgG4. Clin Exp Nephrol 2009;13: particularly if instigated early in the course of the disease. 367–72. Comprehensive, validated diagnostic criteria and a 6 Miyake K, Moriyama M, Aizawa K et al. Peripheral greater understanding of the pathogenesis of IgG4þ CD4þ T cells showing a Th2 phenotype in a MOLPS are required. patient with Mikulicz’s disease associated with lymphadenopathy and pleural effusion. Mod Rheumatol 2008;18:86–90. Rheumatology key message 7 Zen Y, Fujii T, Harada K et al. Th2 and regulatory immune . CSSD-manifesting IgG4þ MOLPS can present with reactions are increased in immunoglobin G4-related SPH and remarkable IgG4-linked IL-4 elevation. sclerosing pancreatitis and cholangitis. Hepatology 2007; 45:1538–46. 8 Ueno K, Watanabe T, Kawata Y et

Journal

RheumatologyOxford University Press

Published: Sep 19, 2010

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