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Wenshuo Zhang, R. Sargis, P. Volden, C. Carmean, X. Sun, M. Brady (2012)PCB 126 and Other Dioxin-Like PCBs Specifically Suppress Hepatic PEPCK Expression via the Aryl Hydrocarbon Receptor
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Curr Envir Health Rpt (2017) 4:439–449 DOI 10.1007/s40572-017-0166-8 SYNTHETIC CHEMICALS AND HEALTH (J HERBSTMAN AND T JAMES-TODD, SECTION EDITORS) Persistent Endocrine-Disrupting Chemicals and Fatty Liver Disease 1 1 1 Andrea L. Deierlein & Sarah Rock & Sally Park Published online: 4 October 2017 Springer International Publishing AG 2017 Abstract sample of gastric bypass surgery patients, examined sex- Purpose of Review Non-alcoholic fatty liver disease specific associations of POPs and liver enzymes, finding ad- (NAFLD) is the most prominent chronic liver disease in verse associations among women only. The noticeable lack of Western countries, affecting approximately 25% of the popu- studies investigating how differences (i.e., biochemical, phys- lation worldwide. Sex-specific differences in the development iological, and behavioral) between men and women may in- of NAFLD are apparent. While obesity and insulin resistance fluence associations of pEDCs and NAFLD represents a large are major contributors to the increasing prevalence of research gap in environmental health. Sexual dimorphism in NAFLD, a growing body of literature suggests that exposure metabolic processes throughout the body, including the liver, to persistent endocrine-disrupting chemicals (pEDCs) may al- is established but often overlooked in the designs and analyses so play a role. This review summarizes recent (2011
Current Environmental Health Reports – Springer Journals
Published: Oct 4, 2017
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