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- Copyright
- © 2009 Wolters Kluwer Health | Lippincott Williams & Wilkins
- ISSN
- 1544-5186
- eISSN
- 1552-2032
- DOI
- 10.1097/01.NME.0000363319.70905.96
- Publisher site
- See Article on Publisher Site
Abstract
wound wise Over the centuries, pressure ulcers have been tective mechanism in the body that di- referred to as decubitus ulcers, bedsores, and lates vessels in the effected area to in- pressure sores. The term pressure ulcer has crease blood flow and speed oxygen to become the preferred name because it most starved tissues. Reactive hyperemia first closely describes the etiology and resultant appears as a bright flush that lasts about ulcer. The National Pressure Ulcer Advisory one-half or three-quarters as long as the Panel (NPUAP) revised its definition of pres- ischemic period. If the applied pressure is sure ulcers at its 2007 consensus conference too high for too long, reactive hyperemia The pressure is to read: “localized injury to the skin and/or fails to meet the demand for blood and on to recognize underlying tissue over a bony prominence, tissue damage occurs. and treat as a result of pressure, or pressure in com- Blanchable erythema can signal immi- bination with shear and/or friction.” nent tissue damage. Erythema results pressure ulcers Pressure ulcers are usually located over from capillary dilation near the skin’s sur- early. bony prominences, such as the sacrum, coc- face. In the patient with a pressure
Journal
Nursing Made Incredibly Easy! – Wolters Kluwer Health
Published: Nov 1, 2009