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Pannexins are new molecular candidates for assembling gap junctions in the cochlea

Pannexins are new molecular candidates for assembling gap junctions in the cochlea AUDITORYAND VESTIBULAR SYSTEMS NEUROREPORT Pannexins are new molecular candidates for assembling gap junctions in the cochlea a a c a,b Wenxue Tang ,Shoeb Ahmad , Valery I. Shestopalov and Xi Lin a b c Departments of Otolaryngology, Cell Biology, Emory University School of Medicine, Atlanta,Georgia and Bascom Palmer Eye Institute, University of Miami, Miami, Florida,USA Correspondence to Xi Lin, PhD, Departments of Otolaryngology and Cell Biology, Emory University School of Medicine, Atlanta,GA 30322,USA Tel: + 404 727 3723; fax: + 404 7276256; e-mail: xlin2@emory.edu Received 2 April 2008; accepted12 May 2008 DOI:10.1097/WNR.0b013e32830891f5 Genetic studies have linked many nonsyndromic deafness patients expressed in the spiral and Scarpa’s ganglion neurons. These data to mutations in genes coding for gap junction proteins. To better for the ¢rst time showed expressions of Panxs in the cochlea, understand molecular identities of gap junctions in the cochlea, therefore adding a new family of gap junction proteins to those we investigated the expression of pannexins (Panxs).Western blot used to form intercellular transport pathways in the cochlea. and reverse transcription-PCR detected the expression of Panx1 NeuroReport 19:1253^1257 2008 Wolters Kluwer Health | and Panx2. Immunolabeling localized Panx1to the inner and outer Lippincott Williams & Wilkins. sulcus, as http://www.deepdyve.com/assets/images/DeepDyve-Logo-lg.png Neuroreport Wolters Kluwer Health

Pannexins are new molecular candidates for assembling gap junctions in the cochlea

Neuroreport , Volume 19 (13) – Aug 1, 2008

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ISSN
0959-4965
eISSN
1473-558X
DOI
10.1097/WNR.0b013e32830891f5
pmid
18695503
Publisher site
See Article on Publisher Site

Abstract

AUDITORYAND VESTIBULAR SYSTEMS NEUROREPORT Pannexins are new molecular candidates for assembling gap junctions in the cochlea a a c a,b Wenxue Tang ,Shoeb Ahmad , Valery I. Shestopalov and Xi Lin a b c Departments of Otolaryngology, Cell Biology, Emory University School of Medicine, Atlanta,Georgia and Bascom Palmer Eye Institute, University of Miami, Miami, Florida,USA Correspondence to Xi Lin, PhD, Departments of Otolaryngology and Cell Biology, Emory University School of Medicine, Atlanta,GA 30322,USA Tel: + 404 727 3723; fax: + 404 7276256; e-mail: xlin2@emory.edu Received 2 April 2008; accepted12 May 2008 DOI:10.1097/WNR.0b013e32830891f5 Genetic studies have linked many nonsyndromic deafness patients expressed in the spiral and Scarpa’s ganglion neurons. These data to mutations in genes coding for gap junction proteins. To better for the ¢rst time showed expressions of Panxs in the cochlea, understand molecular identities of gap junctions in the cochlea, therefore adding a new family of gap junction proteins to those we investigated the expression of pannexins (Panxs).Western blot used to form intercellular transport pathways in the cochlea. and reverse transcription-PCR detected the expression of Panx1 NeuroReport 19:1253^1257 2008 Wolters Kluwer Health | and Panx2. Immunolabeling localized Panx1to the inner and outer Lippincott Williams & Wilkins. sulcus, as

Journal

NeuroreportWolters Kluwer Health

Published: Aug 1, 2008

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