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0360-9294/79/0406-0207$02.00/0 JOURNAL OF THE AMERICAN AUDITORY SOCIETY Vol. 4. No. 6 Copyright 0 1979 by The Williams & Wilkins Co. Printed in U. S. A. PRESBYCUSIS: THE AGING EAR Part 11’ ODED GILAD, M.D. AND ARAM GLORIG, M.D. Ear Research Institute. and the University of Southern California School of Medicine Received October 27, 1978; accepted December 8. 1978 Presbycusis: Vascular Etiology lear vessels. They also observed loss of capillaries and degeneration of hair cells and neural fibers. Vasodilatation is a common treatment for var- They found a strong association between angio- ious inner ear diseases including Meniere’s disease, sclerosis of kidney vessels and angiosclerosis of sudden hearing loss, tinnitus, and presbycusis. The inner ear vessels. Crowe et al. (1934) found arte- dependence of the inner ear on a single end artery riosclerosis in each of his cases with high-fre- without cross-circulation and favorable clinical quency sensorineural hearing loss. Jorgensen re- results in some patients with vasodilatation natu- ported PAS-positive thickening of stria1 vessels in rally leads to speculation about vascular insuffi- all patients (1961b) and in diabetic patients ciency. There is an insufficient amount of histo- (1961~). Johnson and Hawkins (1972) examined pathological evidence of vascular pathology in the
Journal of the American Audiology Society – Wolters Kluwer Health
Published: May 1, 1979
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